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![]() ![]() The aim of this paper is to shed further light at the developmental timing whereby neurological correlates of adverse experiences begin to emerge. In this study we use electroencephalography (EEG) to examine associations of socioeconomic and psychosocial factors with an objective measure of neurofunctional outcomes in 6-month-old infants and 36-month-old children. We previously proposed that these findings likely reflect one of three possibilities: 1) a developmental effect whereby children gradually fall behind on developmental milestones over time due to an acceleration of developmental milestones achieved with age, 2) a cumulative effect whereby exposure to poverty-related risks accumulate over time such that effects only emerge after a certain amount of “environmental exposure” to poverty, or 3) a methodological confound in behavioral assessments whereby the ability of developmental assessments to differentiate between children’s developmental status may improve as children age given the broader range of tasks children can perform as they age. We have also shown that poverty predicts child outcomes via exposure to risks such as poor growth, inflammation, and playful parenting from 27 months onwards, while only playful parenting showed an association with milestone related to language at 6 months ( Jensen et al., 2019c). For example, using data from Bangladesh we have previously shown a pattern whereby children perform as expected with regards to developmental milestones at 6 months, but have dropped significantly at 27 months ( Jensen et al., 2019b). Emerging evidence from longitudinal research suggests that socioeconomic disparities in child development emerge sometime during the first two years of children’s life. Indeed, a large body of literature from both high and low-income countries has shown that children in poor households perform less well on measures of cognitive functions and language compared with wealthier peers ( Jensen et al., 2019b, c Johnson et al., 2016 Rubio-Codina and Grantham-McGregor, 2019). Overall, findings suggest that children’s environment continues to influence the development of EEG oscillations and provide support for the utility of EEG to quantify developmental effects of early life experiences on neural functional outcomes in low income countries.Įnvironmental influences on child development are well-known and behavioral studies from throughout the world show that children living in impoverished circumstances tend to reach developmental milestones later than children living in wealthier families. The finding of a negative relationship of household wealth with beta and gamma power in 36-month-old children differs from findings previously observed in infants in high-income countries. We also find that higher reports of maternal perceived stress are associated with more absolute theta power in frontal and central regions in the 36-month-old children. Among the 36-month-old children, we find that household wealth is negatively associated absolute power in the beta and gamma bands across frontal, central, and parietal electrodes. Among the 6-month-old infants we do not observe any association of household wealth or psychosocial factors with EEG power. ![]() Psychosocial factors include maternal perceived stress and family caregiving activities. Household wealth is assessed as a multi-dimensional composite score encompassing income, assets, and housing materials. In the current paper we explore relationships of spectral EEG power across three regions (frontal, central, and parietal) and four frequency bands (theta, alpha, beta, and gamma) with socioeconomic and psychosocial factors in a cohort of n = 160 6-month-old infants and n = 187 36-month-old children living in Dhaka, Bangladesh. Studies of infants growing up in high-income countries reveal developmental changes in electroencephalography (EEG) power whereby socioeconomic factors - specifically, low SES and low income - are associated with lower EEG power in infants aged newborn to nine months. ![]() ![]()
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![]() ![]() Polina G, Grimson WEL, Shenton ME, Kikinis R (2001) Deformation analysis for shaped based classification, information processing in medical imaging. In: Ninth international conference on medical image understanding and analysis, Bristol, 19–20 July 2005, p 167–170 ![]() Olmos S, Bossa M, Styner M (2005) PCA-based filtering for hypothesis testing on hippocampus shape differences between schizophrenics and normals. Jonke E, Schaefer K, Freudenthaler JW, Prossinger H, Bookstein FL (2003) A cephalometric comparison of skulls from different time periods–the Bronze Age, the 19th century and the present. J Craniofac Surg 18:219–224Įrcan I, Ocakoglu G, Guney I, Yazici B (2008) Adaptation of generalizability theory for inter-rater reliability for landmark localization. Įrcan I, Etoz A, Guney I, Ocakoglu G, Turan Ozdemir S, Kan I, Kahveci R (2007) Statistical shape analysis of nose in Turkish young adults. Houghton-Mifflin/Lahaska Press, Bostonĭryden I (2004) Shapes: statistical shape analysis. In: Erford BT (ed) Assessment for counselors. Forensic Sci Int 159:126–146ĭeQuardo JR, Keshavan MS, Bookstein FL, Bagwell WW, Green WDK, Sweeney JA, Haas GL, Tandon R, Schooler NR, Pettegrew JW (1999) Landmark-based morphometric analysis of first-episode schizophrenia. Proc Natl Acad Sci 95:11406–11411ĭe Greef S, Claes P, Vandermeulen D, Mollemans W, Suetens P, Willems G (2006) Large-scale in vivo Caucasian facial soft tissue thickness database for craniofacial reconstruction. Epilepsia 32:778–782Ĭsernansky JG, Joshi S, Wang L, Haller JW, Gado M, Miller JP, Grenander U, Miller MI (1998) Hippocampal morphometry in schizophrenia by high dimensional brain mapping. Teratology 64:4–32Ĭavalheiro EA, Leite JP, Bortolotto ZA, Turski WA, Ikonomidou C, Turski L (1991) Long-term effect of pilocarpine in rats: structural damage of the brain triggers kindling and spontaneous recurrent seizures. Cambridge University Press, New Yorkīookstein FL, Sampson PD, Streissguth AP, Connor PD (2001) Geometric morphometrics of corpus callosum and subcortical structures in the fetal-alcohol-affected brain. IEEE Trans Pattern Anal Machine Intell 11:567–585īookstein FL (1991) Morphometrics tools for landmark data. Hippocampus 11:452–468īookstein FL (1989) Principal warps: thin-plate splines and the decomposition of deformations. J Anat 206:373–385Īndre V, Marescaux C, Nehlig A, Fritschy JM (2001) Alterations of hippocampal gabaergic system contribute to development of spontaneous recurrent seizures in the rat lithium pilocarpine model of temporal lobe epilepsy. Anat Rec 285:690–698Īldridge K, Kane AA, Marsh JL, Yan P, Govier D, Richtsmeier JT (2005b) Relationship of brain and skull in pre- and postoperative sagittal synostosis. J Anat 201:31–39Īldridge K, Kane AA, Marsh JL, Panchal J, Boyadjiev SA, Yan P, Govier D, Ahmad W, Richtsmeier JT (2005a) Brain morphology in nonsyndromic unicoronal craniosynostosis. ![]() Sage, LondonĪldridge K, Marsh JL, Govier D, Richtsmeier JT (2002) Central nervous system phenotypes in craniosynostosis. This study may serve as an initial reference for future studies on shape alteration of the hippocampus associated with certain medical conditions.Īldenderfer MS, Blashfield RK (1989) Cluster analysis. In conclusion, our landmark-based methodology detected regional differences in the hippocampus in epilepsy. The results strongly indicated that the normalized hippocampal shape of the epileptic group was different from the nonepileptic group deformation was seen most significantly in the medial regions of the cornu ammonis (CA1 and CA3) of the hippocampus. Nineteen rat brains (ten healthy and nine epileptic) with hematoxylin and eosin images of the hippocampus were used. Statistical shape analysis is of increasing interest to the neuroimaging community because of its potential for locating morphological changes. We have used the statistical shape analysis method to illustrate hippocampal shape deformation due to epilepsy. In this study, we aimed to (1) propose landmarks for the hippocampus in a rat brain using an experimental study and (2) investigate hippocampus shape changes in a rat brain with epilepsy using the statistical shape analysis method. ![]() ![]() ![]() You can save programmes as a favourite and pick up on a programme where you last viewed. You can browse programmes by name, category, date and latest. Social sharing features allow you to share programmes. The RTÉ Player App supports Chromecast so you can cast to your big screen using your Smart TV. Search for the title you want to watch in our Find Section. 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